Short answer: most evidence suggests that common lysergamide analogs like 1P-LSD, ALD-52, 1cP-LSD, and 1B-LSD act mainly as prodrugs of LSD. In animals, enzymes remove the acyl group and release LSD. Limited human data with 1P-LSD shows it appears in blood briefly while LSD becomes the dominant compound.
What users often notice aligns with that: onset can feel slightly slower for some, but once converted, the effects and time course are reported as very similar to LSD. Individual responses vary, and human research is still limited.
On potency, these analogs are roughly equipotent to LSD after correcting for molecular weight. By weight they may need about 15 to 20 percent more to match LSD, though formulation and metabolism can shift this.
Legal status differs by country and can change, and I cannot verify your local situation. For a concise overview that compares LSD and its analogs, see this summary: how do LSD analogs compare to LSD.
Hope this clarifies the core differences.