Short answer: most popular lysergamide analogs such as 1P-LSD, ALD-52, 1B-LSD, and 1cP-LSD appear to act mainly as prodrugs of LSD, so their core effects are largely mediated by LSD and are not fundamentally different.
Evidence from animal work shows rapid enzymatic removal of the acyl group, releasing LSD. Human pharmacokinetic data indicate 1P-LSD appears briefly in serum, then declines as LSD rises and persists. This aligns with reports of a slightly slower onset but a largely similar peak experience.
When corrected for molecular weight, lab assays find comparable potency and LSD-like behavioral profiles across these analogs. Individual differences, product purity, and context can still shift timing and intensity.
Legal status varies by location and can change; I cannot verify the current situation in your area. For a concise overview of how these analogs compare with LSD, see this Q&A.
Hope this helps.